Postinflammatory hyperpigmentation
Postinflammatory hyperpigmentation هڪ چمڙي جي حالت آهي جنهن جي خاصيت چمڙي جي سوزش کان پوءِ رنگن جي وڌ سان ٿيندي آهي. Postinflammatory hyperpigmentation سج جي ڊگهي نمائش، سوزش، يا چمڙي جي ٻين زخمن جي ڪري ٿي سگهي ٿو، جن ۾ مَن سان لاڳاپيل آهن. اونداهي رنگ جي چمڙي وارا ماڻهو اڪثر ڪري وڌيڪ سج جي نمائش سان هائپرپيگمينٽيشن جو شڪار ٿيندا آهن.
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ReferencesPostinflammatory hyperpigmentation (PIH) هڪ بار بار چمڙي جو مسئلو آهي جيڪو چمڙي جي سوزش يا زخم کان پوءِ ٿئي ٿو. اهو ڊگهو وقت تائين هلندو رھي ٿو ۽ اونداهي چمڙي جي ٽون ۾ وڌيڪ واضح آهي (Fitzpatrick skin types III–VI). جيتوڻيڪ اهو اڪثر پاڻ ۾ بهتر ٿي ويندو آهي، پر ڪجه وقت وٺي سگهي ٿو، تنهنڪري علاج گهڻو وقت گهربل آهي. مختلف علاجن کي گڏ ڪرڻ بهتر نتيجا ڏيندو آهي.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation چمڙي جي سوزش جو عام نتيجو آهي. اهو اونداهي رنگ جي چمڙي وارن ماڻهن کي وڌيڪ سخت ۽ بار بار متاثر ڪري ٿو. مطالعي مان معلوم ٿئي ٿو ته Postinflammatory hyperpigmentation جھڙا مسئلا بنيادي سببن مان آهن، ڇو ته ڪاري چمڙي جي ٽون وارا ماڻهو ڊرماتولوجيڪل سنڀال ڳوليندا آهن. ابتدائي علاج Postinflammatory hyperpigmentation کي حل ڪرڻ لاءِ اهم آهي ۽ عام طور تي سوزش واري حالت کي منظم ڪرڻ سان شروع ٿئي ٿو. علاج جي پهرين لائن ۾ عام طور تي ٽاپيڪل ايجنٽ شامل آهن جيڪي چمڙي کي روشن ڪن ٿا، ۽ انهن کي سن اسڪرين سان گڏ استعمال ڪرڻ گهرجي. اهي ايجنٽ، جهڙوڪ hydroquinone, azelaic acid, kojic acid, arbutin, licorice extracts، مؤثر طور تي اضافي رنگن کي گهٽائي سگهن ٿا. اضافي طور تي retinoids, mequinol, ascorbic acid, niacinamide, N‑acetyl glucosamine, soy پڻ depigmenting ايجنٽ طور استعمال ڪيا ويا آهن. جڏهن ته بنيادي علاج عام طور تي سطحي هائپرپيگمينٽيشن لاءِ اثرائتو هوندا آهن، سخت ڪيسن ۾ (laser, chemical peel) جهڙا طريقا ضروري ٿي سگهن ٿا. Postinflammatory hyperpigmentation جي جلن ۽ خراب ٿيڻ کان بچڻ لاءِ انهن علاجن سان احتياط ڪرڻ ضروري آهي.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
