Postinflammatory hyperpigmentation
Postinflammatory hyperpigmentation هڪ چمڙي جي حالت آهي جنهن جي خاصيت چمڙي جي سوزش کان پوءِ چمڙي ۾ رنگن جي واڌ سان ٿيندي آهي. Postinflammatory hyperpigmentation سج جي ڊگھي نمائش، سوزش، يا چمڙي جي ٻين زخمن جي ڪري ٿي سگھي ٿو، جن ۾ مَن سان لاڳاپيل آھن. اونداهي چمڙي جا ماڻهو اڪثر ڪري وڌيڪ سج جي نمائش سان هائپر پيگمينٽيشن جو شڪار هوندا آهن.
☆ 2022 ۾ جرمني مان Stiftung Warentest نتيجن، ModelDerm سان صارفين جي اطمينان صرف ادا ڪيل ٽيلي ميڊيسن مشوري جي ڀيٽ ۾ ٿورو گهٽ هئي.
References
Postinflammatory hyperpigmentation (PIH) هڪ بار بار چمڙي جو مسئلو آهي جيڪو چمڙي جي سوزش يا زخم کان پوء ٿئي ٿو. اهو هڪ ڊگهو وقت تائين هلندو آهي ۽ ماڻهن ۾ اونداهي چمڙي جي ٽون سان وڌيڪ خراب آهي (Fitzpatrick skin types III–VI) . جيتوڻيڪ اهو اڪثر پاڻ تي بهتر ٿي ويندو آهي، اهو ڪجهه وقت وٺي سگهي ٿو، تنهنڪري علاج اڪثر وقت تائين گهربل آهي. مختلف علاجن کي گڏ ڪرڻ بهترين ڪم ڪري ٿو.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation چمڙي جي سوزش جو هڪ عام تسلسل آهي. اهو اونداهي چمڙي وارن ماڻهن کي وڌيڪ سخت ۽ بار بار متاثر ڪري ٿو. مطالعي مان معلوم ٿئي ٿو ته postinflammatory hyperpigmentation جھڙا مسئلا بنيادي سببن مان آھن ڇو ته ڪارو چمڙي جي ٽون وارا ماڻھو ڊرماتولوجيڪل سنڀال ڳوليندا آھن. ابتدائي علاج postinflammatory hyperpigmentation کي حل ڪرڻ لاءِ اهم آهي ۽ عام طور تي ابتدائي سوزش واري حالت کي منظم ڪرڻ سان شروع ٿئي ٿو. علاج جي پهرين لائن ۾ عام طور تي شامل آهي ٽوپيل ايجنٽ استعمال ڪرڻ جيڪي چمڙي کي روشن ڪن ٿا حفاظت لاءِ سن اسڪرين سان گڏ. اهي ايجنٽ، جهڙوڪ hydroquinone, azelaic acid, kojic acid, arbutin, licorice extracts ، مؤثر طور تي اضافي رنگن کي گھٽائي سگهن ٿا. اضافي طور تي، retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, soy پڻ استعمال ڪيا ويا آهن depigmenting ايجنٽ، نئين علاج سان گڏ. جڏهن ته بنيادي علاج عام طور تي سطحي سطح جي هائپر پيگمينٽيشن لاءِ اثرائتو هوندا آهن، ضعيف ڪيسن لاءِ طريقا (laser, chemical peel) ضروري هوندا. Postinflammatory hyperpigmentation جي جلن ۽ خراب ٿيڻ کان بچڻ لاءِ انهن علاجن سان احتياط ڪرڻ ضروري آهي.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.
